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Clinicopathological features of nine cases of non-cirrhotic portal hypertension 100%

Clinical and biochemical data and the alterations in livers resected at transplantation (n ¼ 7) or at autopsy (n ¼ 2) were gathered in five males and four females (ages 15–78 years) without aetiological factors for chronic hepatic disease who had oesophageal varices and splenomegaly in the absence of typical cirrhosis.


noncirrhotic portal hypertension[1] 96%

In general, the lesions in NCPH are vascular in nature and can be classified based on the site of resistance to blood flow as ‘‘prehepatic,’’ ‘‘hepatic,’’ and ‘‘posthepatic.’’ The ‘‘hepatic’’ causes of NCPH can be subdivided into ‘‘presinusoidal,’’ ‘‘sinusoidal,’’ and ‘‘postsinusoidal’’ (Box 1).


New assessment of hepatic encephalopathy 2011 87%

Review New assessment of hepatic encephalopathy Juan Córdoba⇑ Servei de Medicina Interna-Hepatologia, Hospital Vall d’Hebron, Universitat Autònoma de Barcelona and Centro de Investigación Biomédica en Red de Enfermedades Hepáticas y Digestivas (CIBEREHD), Paseo Vall d’Hebron 119, Barcelona 08035, Spain Hepatic encephalopathy (HE) is a common complication of cirrhosis that requires careful appraisal of the clinical manifestations, evaluation of the underlying neurological disorders, and assessment of liver function and the portal-systemic circulation.


A diagnostic approach to hyperferritinemia 84%

We used observations from a large series of patients with hepatic iron overload documented by liver iron concentration measurement from two referral practices as a gold standard to guide the interpretation of the predictive values of non-invasive iron tests.


Non cirrhotic portal hypertension versus idiopathic portal hypertension 83%

Other causes include schistosomiasis, hepatic venous outflow tract obstruction, veno-occlusive disease and congenital hepatic fibrosis.


RASOANAIVO efficacity of herbal antimalarial 79%

Philippe Rasoanaivo Head of malaria projects, IMRA Head of RITAM pre-clinical group There is a great potential of antimalarial treatments in different components of biodiversity BIODIVERSITY Ecosystem diversity Ecochemistry Species diversity Chemodiversity Antimalarial treatments Culture diversity Ethnobotany Critical evaluation of the present state of knowledge Lack of clinical observational studies on the effectiveness of reputed antimalarial treatments Insufficient work dedicated to the antiplasmodial screening of plants as well as bioassay-guided fractionations of active extracts Lack of antimalarial screening on the hepatic stage of malaria parasites The way forward Strengthening the search for active constituents of antimalarial plants in the erythrocytic stage Extending antiplasmodial screening to other endemic plants to encompass large structure diversity Extending the screening of antimalarial plants to the hepatic stage Performing clinical observational studies on reputed antimalarial plants for home management of malaria (HMM) in community-based approach World malaria distribution and occurrence of antimalarials Artemisinin Quinine ?


Article SP Académie de Chirurgie 78%

hepatic function.


Postdoc Lifesearch WP1 EN 2013 75%

Indeed, HDL-C has the unique ability to excreted cholesterol excess out of the body through hepatic biliary lipid secretions.


Resume 62%

files management, customer relations, materials order, finding technical solutions - In charge of the components design via Solidworks Microsoft office Key skills 8 rue Martin Basse 69300 Caluire, France 04-78-23-10-76 06-88-73-87-53 2012 (2 mois) Croix-Rousse University Hospital - Hepatic surgery Lyon, France Temporary job :


Chen 2006 60%

Two isoforms of MAO, types A and B, have been described and differ with respect to localization and substrate specificity (Table 1).21-23 The gastrointestinal and hepatic MAO-A isoform plays a crucial role in deactivating circulating catecholamines and dietary sympathomimetics (eg, tyramine), whereas brain MAO-A contributes to oxidative deamination of DA, norepinephrine, and serotonin.


International-Medical-Research-School 59%

-Renal Ischemia. -Hepatic ischemia -Overactomy.


Aromatherapy-QuickStudy 56%

these are synthetic, have no therapeutic properties, and should never be used in aromatherapy ■ Tip In order to be effective, essential oils must be of the highest quality Storing Essential Oils ■ ■ ■ ■ ■ Use of essential oils with babies and children requires different procedures than those covered in this guide 1 ■ Store in tightly closed, dark or opaque glass jars Keep away from heat, moisture and direct light Open bottles only for use and close again as soon as possible Do not refrigerate The shelf life of most essential oils is about 12 to 18 months (when properly stored) Citrus oils and juniper oil generally have a shelf life of about 6 months (when properly stored) Essential Oils Frankicense Bergamot 7 Botanical Name Citrus bergamia Actions analgesic, antiseptic, antispasmodic, calmative, carminative, diuretic, stomachic, uplifting Uses acne, anxiety, colds/flu, cold sores, cystitis, depression, flatulence, stomachache, stress Cautions/Comments phototoxic* 1 Botanical Name Boswellia carteri Actions analgesic, antidepressant, anti-inflammatory, astringent, calmative, cicatrizant, cytophylactic, diuretic, emmenagogue, expectorant, strengthening, tonic, uterine, vulnerary Uses anxiety, bronchitis, colds/flu, coughs, depression, dysmenorrhea, laryngitis, mature skin, meditation aid, menorrhagia, nervous exhaustion, scars, slack skin, stress Cedarwood, Atlas Botanical Name Cedrus atlantica Actions antifungal, antiseptic, aphrodisiac, astringent, calmative, diuretic (mild), expectorant, mucolytic, tonic Uses acne, arthritis, bronchitis, cystitis, depression, fungal infections, oily skin, rheumatism, stress Cautions/Comments avoid during pregnancy 2 Geranium Botanical Name Pelargonium graveolens Actions analgesic, antidepressant, anti-inflammatory, astringent, cicatrizant, cytophylactic, diuretic, hemostatic, lymphatic stimulant, tonic, vasoconstrictor, vulnerary Uses acne, bruises, depression (mild), fluid retention, menopause, nervous tension, neuralgia, PMS, scars, skin care (all types), sore throat, stress Cautions/Comments may cause wakefulness if used at night 8 Chamomile, Roman 3 Botanical Name Anthemis nobilis Actions analgesic, antidepressant, anti-inflammatory, antineuralgic, antiseptic, calmative, carminative, digestive, diuretic, emmenagogue, febrifuge, hepatic, muscle relaxant, nervine, tonic, vulnerary Uses anxiety, arthritis, dry skin, flatulence, headaches, immune system, indigestion, insomnia, menstrual cramps, muscle pain, neuralgia, PMS, rheumatism, sprains, stress Cautions/Comments may cause drowsiness;


liver 56%

reticular fiber network in the case of hepatic peliosis Fungal infection Routine staining Amyloidoses of any type, in particular cardiovascular Sediments in renal tubules and vascular walls following ethylene glycol intoxication Myelin sheath staining Connective tissue stain, for example, in the case of liver cirrhosis Hyaline fibrin thrombi in the case of shock Hematopoietic marrow, differentiation of cells of the myeloid and lymphatic line;


Étiologies du syndrome d’hypertension portale en milieu tropical 55%

— a transparietal hepatic puncture biopsy with the 1.4 mm diameter Menghini’s needle.


biblio covid19 4 52%

- respiratory, cardiac, hemorrhage, hepatic, and renal damage were found in 100%, 89%, 80.5%, 78.0%, and 31.7% of patients - Creatinine >133μmol/L ;


Lustig 2012 The toxic truth about sugar 48%

Chronic ethanol exposure Chronic fructose exposure Haematological disorders Electrolyte abnormalities Hypertension Hypertension (uric acid) Cardiac dilatation Cardiomyopathy Myocardial infarction (dyslipidaemia, insulin resistance) Dyslipidaemia Dyslipidaemia (de novo lipogenesis) Pancreatitis Pancreatitis (hypertriglyceridaemia) Obesity (insulin resistance) Obesity (insulin resistance) Malnutrition Malnutrition (obesity) Hepatic dysfunction (alcoholic steatohepatitis) Hepatic dysfunction (non-alcoholic steatohepatitis) Fetal alcohol syndrome Addiction Habituation, if not addiction Source:


Lustig Nature 48%

Chronic ethanol exposure Chronic fructose exposure Haematological disorders Electrolyte abnormalities Hypertension Hypertension (uric acid) Cardiac dilatation Cardiomyopathy Myocardial infarction (dyslipidaemia, insulin resistance) Dyslipidaemia Dyslipidaemia (de novo lipogenesis) Pancreatitis Pancreatitis (hypertriglyceridaemia) Obesity (insulin resistance) Obesity (insulin resistance) Malnutrition Malnutrition (obesity) Hepatic dysfunction (alcoholic steatohepatitis) Hepatic dysfunction (non-alcoholic steatohepatitis) Fetal alcohol syndrome Addiction Habituation, if not addiction Source:


brmedj02298-0063c 46%

retention and microscopical evidence of liver disease in many of his cases, and recently hepatic dysfunction has been demonstrated in Felty's syndrome (17 January, p.


Article-PUGG-AVK-Drugs-and-Aging-Octobre-2013 45%

8.7 (3.2) Associated medications [% (no.)] 51.5 (1,356) Dementia 53.4 (1,399) Heart failure 43.9 (1,150) Depression 36.2 (950) Stroke 33.1 (872) C2 falls during the past year 23.7 (619) Peripheral artery disease 18.6 (486) Diabetes mellitus 18.2 (479) Renal failurea CLCR [60 mL/min 24.6 (601) CLCR [30 and B60 mL/min 59.3 (1,469) CLCR B30 mL/min 16.4 (407) Myocardial infarction 14.1 (369) Cancer 10.3 (272) Gastroduodenal ulcer Chronic hepatic disease 9.3 (243) 2.7 (71) CLCR creatinine clearance, SD standard deviation a CLCR derived from the Cockroft–Gault formula patients who spent less than 100 % of TTR, INR values were more often below the therapeutic target than above (first INR test:


Résumé Plathelminthes 45%

Préparé par M. SOUTTOU K.


nejmra1206531 42%

The risk is influenced by the type, number, and location of valvular prostheses, as well as by the presence or absence of associated heart failure, atrial fibrillation, history of thromboembolism, and intracardiac thrombi.19,20 In patients with venous thromboembolism, the risks of recurrent thrombosis, thrombus propagation, and embolization are elevated for 3 months after the diagnosis and initiation of anticoagulation therapy.21 The risk of recurrence differs depending on whether the venous thromboembolism was provoked (in which case the risk decreases with resolution of the underlying risk factor) or unprovoked (i.e., idiopathic) (Table 2).22 CANCER Patients with cancer have an increased risk of periprocedural thrombosis owing to cancer-specific prothrombotic activity, hormonal therapy, angiogenesis inhibitors, radiotherapy, and the presence of indwelling central venous catheters.23 Concurrently, there is an increased risk of bleeding24 because of the administration of prophylactic agents for the prevention of venous thromboembolism, chemotherapy-related hepatic and renal dysfunction and thrombocytopenia, and tumor friability.


RMS La cardiomyopathie cirrhotique 41%

Relevance of hepatic venous pressure gradient and serum calcium.


Am. J. Epidemiol.-1996-Ferrini-642-4 35%

Because sex hormone-binding globulin is produced in the liver, the results suggest a caffeine effect on hepatic metabolism.