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ADHD Diagnostic Flowchart 100%

Assess no yes Does the young person have significant personal and social impairment Exit no yes Do the problems include severe developmentally inappropriate inattention, impulsivity and hyperactivity that impairs functioning?


bisphenol et hyperactivité (1) 97%

Kurraschb,c,1 Departments of aBiological Sciences and bMedical Genetics and cAlberta Children’s Hospital Research Institute, University of Calgary, Calgary, AB, Canada T2N 4N1 Bisphenol A (BPA), a ubiquitous endocrine disruptor that is present in many household products, has been linked to obesity, cancer, and, most relevant here, childhood neurological disorders such as anxiety and hyperactivity.


Mental Health alu et autre métaux 84%

Many of the tables in this document provide information about likely places of exposure to toxic substances, including at home, at school, in the Learning and Developmental Disabilities Initiative repetitive behaviors or mental acts intense fear, helplessness, or horror recurrent and intrusive images, thoughts, distressing dreams, feeling as if a traumatic event is recurring symptoms of increased arousal  difficulty falling or staying asleep  irritability or outbursts of anger  difficulty concentrating  hypervigilance  exaggerated startle response Attention-Deficit and Disruptive Behavior Disorders includes attention-deficit/hyperactivity, conduct and oppositional defiant disorders inattention hyperactivity impulsivity aggression to people and animals destruction of property deceitfulness or theft serious violation of rules negativistic, hostile and defiant behavior Psychotic Disorders includes schizophrenia and other psychotic disorders delusions hallucinations disorganized speech grossly disorganized or catatonic behavior flat or inappropriate affect workplace, in food and water, and so on.


trouble-de-lattention-de-lenfant note de cadrage 81%

Abréviations les plus courantes Abréviation Libellé ADHD Attention deficit-hyperactivity disorder AMM Autorisation de Mise sur le Marché ANAES Agence Nationale d’Accréditation et d’Evaluation de la Santé CADDRA Canadian Attention Deficit Hyperactivity Disorder Resource Alliance CAMSP Centre d’Action Médico-Sociale Précoce CBCL Child Behavior CheckList CFTMEA Classification française des troubles mentaux de l’enfant et de l’adolescent CIM 10 Classification internationale des maladies 10e révision CMP Centre Médico-Psychologique CPT Continuous Performance Test DGS Direction Générale de la Santé DSM IV TR Diagnostic and Statistical Manual of Mental Disorders version IV texte révisé ECG Electrocardiogramme INSERM Institut national de la santé et de la recherche médicale LP Libération Prolongée MDPH Maisons Départementales des Personnes Handicapées OMS Organisation mondiale de la Santé PAI Projet d’Accueil Individualisé PPRE Programme personnalisé de réussite éducative Sros Schémas régionaux d’organisation sanitaire TDAH Trouble Déficit de l’Attention Hyperactivité TOC Trouble Obsessionnel Compulsif WSCT Wisconsin Sorting Card Test HAS - Service des bonnes pratiques professionnelles – Décembre 2012 4/33 Note de cadrage - Conduite à tenir devant un enfant ou un adolescent ayant un déficit de l'attention et/ou un problème d'agitation 1.


TDAH prise en charge et géneralisation des compétences 79%

Effects of reinforcement schedule and task difficulty on the performance of attention-deficit hyperactivity disordered and control boys.


7-Spielewoy-Psychopharm 2001 79%

Mice lacking the dopamine transporter (DAT–/–) exhibit high extracellular dopamine levels and marked hyperactivity.


TDAH, quels liens avec l’attachement 77%

HPA axis 257 Summary Attention-deficit/hyperactivity disorder (ADHD) is defined on the basis of developmentally inappropriate inattention, motor activity and impulsivity that emerges early in development and causes impairment in social and academic functioning.


Chesney et al-2014-World Psychiatry 73%

mortality OR death OR suicide OR suicidal OR suicidality, review OR meta-analysis OR meta-analytic, psychiatry OR psychiatric OR mental OR mood OR affective OR depression OR depressive OR dysthymia OR cyclothymia OR adjustment OR anxiety OR anxious OR “obsessive compulsive” OR 153 Figure 1 Flow chart of systematic search strategy OCD OR panic OR “post-traumatic” OR posttraumatic OR PTSD OR neurosis OR neuroses OR bipolar OR manic OR schizophrenia OR psychotic OR psychosis OR psychoses OR dementia OR demented OR Alzheimer OR “learning disability” OR “learning disabilities” OR IQ OR “mental retardation” OR autism OR autistic OR Asperger OR “attention deficit” OR ADHD OR hyperactivity OR hyperkinetic OR conduct OR disruptive OR personality OR personalities OR borderline OR antisocial OR psychopathic OR dissocial OR forensic OR narcissistic OR schizoid OR schizotypal OR paranoid OR dependent OR avoidant OR “emotionally unstable” OR eating OR anorexia OR bulimia OR EDNOS OR heroin OR opioid OR opioids OR cocaine OR cannabis OR marijuana OR alcohol OR alcoholism OR benzodiazepine OR benzodiazepines OR hypnotic OR hypnotics OR amphetamine OR amphetamines OR barbiturate OR barbiturates OR smoking OR smokers OR cigarette OR cigarettes’.


Vit D Levels in Recurrent Respiratory Chronic cough 73%

Inflammatory processes of the respiratory tract are associated with up regulation of both cough hyperactivity and bronchial hyperactivity and may lead to asthma or are associated with chronic cough.


8-Mead-EJN 2002 60%

However, interpretation of these results is complicated due to the basal hyperactivity of DAT-KO mice, as the effects of stimulants were studied from different baseline levels of activity;


aut01 60%

Such stories help distinguish between AS and AttentionDeficit/Hyperactivity Disorder, a common presenting 123 Appl Psychophysiol Biofeedback (2010) 35:63–81 diagnosis.


The Online Dog Trainer 56%

Issues With Hyperactivity 25. Excessive Licking or Chewing 26.


First Excerpt Spirit Made Smaller 49%

At the last parent-teacher conference, Graywood was informed his child demonstrated signs of attention deficit hyperactivity disorder.


weismann-arcache 2012 44%

Tordjman) for problems at school and psychoaffective (depression and/or anxiety disorders) and behavioral (hyperactivity and/or aggressive conduct) difficulties, with a control group who originally came to a private practice for a psychological assessment (carried out by C.


fr caddraGuidelines2011InformationResources 42%

Children and Adults with Attention Deficit Hyperactivity Disorder (site en anglais seulement)


prescott 2009 34%

DBS appears to mimic the effect of beneficial lesions instead of exacerbating the hyperactivity in the basal ganglia output neurons, but despite the discernible clinical benefits of STN DBS, its mechanism of action remains unclear.


dmn ca 33%

Altered activation of the default network has been reported in functional imaging studies of patients with neuropsychiatric disorders such as dementia, schizophrenia, epilepsy, anxiety and depression, autism and attention deficit/hyperactivity disorder (Buckner et al., 2008;


epigenetic et trauma 32%

published online 13 December 2011 Introduction Exposure to early life adverse events, including abuses and neglect, significantly increases the risk of psychopathology in adulthood.1,2 In humans as well as in animals, response to stress, and particularly to childhood trauma, is mediated by the hypothalamic–pituitary–adrenal (HPA) axis, which includes release of corticotrophin-releasing factor and secondarily results in secretion of glucocorticoids.3 Early life stress has been shown to induce persistent changes in corticotrophin-releasing factor neurotransmission and sustains alteration of the HPA axis.4,5 Hyperactivity as well as decreased activity of the HPA axis in response to stress has been demonstrated in humans with a history of childhood maltreatment.4–6 It has been speculated that the direction of such HPA alterations may depend on the characteristics of the stressor such as the type, duration, intensity or timing.7,8 Nevertheless, although the main role of this HPA axis is to prevent over-response to stress,9 prolonged exposure to elevated levels of glucocorticoids has damaging effects on the developing brain, which may secondarily lead to behavioral problems later in life.10–13 Recent studies point to the epigenetic mechanisms as a process by which environmental factors may alter the gene expression involved in control of the HPA axis.


guenole 2013 31%

i.e., acted-out problems, such as hyperactivity, irritability, rule breaking, or belligerence, which denote undercontrol of behavior and emotion) scores.


1505.full 30%

Exclusionary criteria were the use of psychotropic medications or diagnosis of a psychiatric disorder other than attention deficit hyperactivity disorder.


effects on neuron 29%

Perinatal exposure to nicotine is a known risk factor for sudden infant death syndrome [38], low-birth-weight infants [39], and attention deficit/hyperactivity disorder [40].


ioi30213 29%

(1) Autonomic hyperactivity (eg, sweating or pulse rate ⬎100/min) (2) Increased hand tremor (3) Insomnia (4) Nausea or vomiting (5) Transient visual, tactile, or auditory hallucinations or illusions (6) Psychomotor agitation (7) Anxiety (8) Grand mal seizures C.


TabacGrossesse 28%

however, the underlying mechanisms specific to smoking in humans are not well understood.3,8 It has been suggested that anorexigenic, hypoxic, vascular, and placental effects of nicotine may have direct teratogenic influences on the fetus and result in adverse physiological and psychological development.9 Longitudinal epidemiological studies have reported statistical associations between the extent of maternal smoking during pregnancy and subsequent offspring conduct disorder,10-14 attention-deficit/hyperactivity disorder,15,16 and criminal behavior.17,18 Some investigations have provided evidence of a dose-response relationship between the number of cigarettes smoked during pregnancy and the rate of subsequent conduct problems in offspring.19 However, the effect of maternal pregnancy smoking on offspring conduct problems can be confounded by several background factors, including race/ethnicity, early age at pregnancy, low socioeconomic status, child-rearing environment, and history of maternal psychopathologic conditions.11,13,20-23 For example, mothers who smoke during pregnancy are more likely to provide a child-rearing environment that promotes or at least condones externalizing behavior.21 Therefore, the postnatal environment (independent of pregnancy smoking) may influence the development of conduct problems.