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Réponses pour «serotonin»:

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RapportMD 100%

serotonin, dopamine, and norepinephrine.5 Like other amphetamines, MDMA causes these neurotransmitters to be released from their storage sites in neurons, resulting in increased neurotransmitter activity.


6-Hall-Neurosci-2002 94%

UHLa a b Molecular Neurobiology Branch, NIDA-IRP, NIH, Box 5180, Baltimore, MD 21224, USA Deptartments of Cell Biology and Medicine and HHMI, Duke University, Durham, NC 27710, USA c Department of Psychiatry, University of Wu«rzburg, 97080 Wu«rzburg, Germany d Laboratory of Clinical Science, NIMH-IRP, NIH, Bethesda, MD 20892, USA Abstract;Cocaine blocks uptake by neuronal plasma membrane transporters for dopamine, serotonin and norepinephrine, producing subjective e¡ects in humans that are both euphoric/rewarding and also fearful, jittery and aversive.


5-Sora-PNAS-2001 93%

Combined dopamine and serotonin transporter knockouts eliminate cocaine place preference Ichiro Sora*, F.


Chen 2006 87%

Physiologically, the role of MAO is to catalyze the oxidative deamination of a variety of endogenous neurotransmitters, such as dopamine (DA), epinephrine, norepinephrine, and serotonin, as well as to detoxify exogenous monoamines, such as tyramine.


AlfabetoEmocionalSARD-B 84%

Alfabeto emocional El Dr.


cocaine.piperidine-based.analogs.sar-2 83%

Potent Dopamine and Serotonin Reuptake Inhibitors Amir P.


GuidelinesAnxiety 80%

First-line pharmacological treatments for these disorders are selective serotonin reuptake inhibitors (for all disorders), serotonin-norepinephrine reuptake inhibitors (for some) and pregabalin (for generalized anxiety disorder only).


de bodinat Agomelatine 2010 79%

In their wake, the 1980s and the 1990s witnessed the introduction of more specific inhibitors of serotonin (also known as 5-hydroxytryptamine;


3-Sora-PNAS-98 78%

Conditioned place preference can be established in dopamine- and in serotonin-transporter knockout mice ICHIRO SORA*, CHRISTINE WICHEMS†, NOBUYUKI TAKAHASHI*, XIAO-FEI LI*, ZHIZHEN ZENG*, RANDAL REVAY*, K LAUS-PETER LESCH‡, DENNIS L.


MIA cardio ECN132 infarctus myocard 78%

LECLERQ SCA avec sus décalage de ST ou mort subite cardiaque mechanical effect Vasoactive effect Distal Distal embolisation embolisation Platelet Platelet content content release release (serotonin….) (serotonin….) Blocage Blocage mécanique mécanique Vasoconstriction Vasoconstriction © LIPCOM Faculté de Médecine Montpellier-Nîmes 2e cycle – MIA – Cardiologie – item 132 - L’infarctus du myocarde Année Universitaire 2007 - 2008 La plaque fragile ou vulnérable:


2-Rocha-Nat1998 76%

These results demonstrate that the interaction of cocaine with targets other than DAT, possibly the serotonin transporter, can initiate and sustain cocaine self-administration in these mice.


Building-Alien-Worlds-The-Neuropsychological-and-Evolutionary-Implications-of-the-Astonishing-Psychoactive-Effects-of-N-N-Dimethyltryptamine-DMT 74%

However, by regarding this unique molecule as equivalent to serotonin, an endogenous neuromodulator with a long-standing relationship with the brain, DMT’s effects may be explained.


Antidepressants- Marketing & Lobbying & PR 74%

is a woody climbing shrub, able to increase naturally the serotonin to get a better mood and a better quality of sleep.


7-Spielewoy-Psychopharm 2001 73%

In addition, we tested the implication of the serotonin system in the observed effects.


Serotonin étiquette 66%

Serotonin 8,5° IBU EBC FG OH H2N NH Ingrédients :


DNS 63%


Diagnosis and management of Raynaud's Sd 62%

Serotonin reuptake inhibitors—The exact role of serotonin reuptake inhibitors in the treatment of Raynaud’s phenomenon is not yet clear.


Article-PUGG-AVK-Drugs-and-Aging-Octobre-2013 60%

age, gender, the type of care structure, characteristics of the VKA treatment (indication for VKA, type of VKA, time since institution of VKA, results and dates of the last two INRs, history of INR [4.5, and history of major bleeding), current co-medications known to interact with VKA [aspirin, clopidogrel, NSAIDs, statins (HMG-CoA reductase inhibitors), antibiotics, antifungal drugs, proton pump inhibitors, selective serotonin reuptake inhibitors (SSRIs), and acetaminophen], and co-morbidities (Charlson’s score [28]).



For example, in marine snail Aplysia, in the absence of a neuron-specific ApCPEB, serotonin mediated enhancement of synaptic transmission fails to persist beyond 24 h [7,10].


Central fatigue and neurotransmitters, can thermoregulation be manipulated 56%

It is clear that different brain neurotransmitters – such as serotonin, dopamine and noradrenaline – are implicated in the occurrence of fatigue, but manipulation of these neurotransmitters produced no conclusive results on performance in normal ambient temperature.