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hyperthyroidism and pulmonary (4) 100%

Most reports have focused on only the venous thromboembolism risk, and few of them have studied specifically the association between hyperthyroidism and pulmonary embolism (PE).


hyperthyroidism-and-pulmonary-4 100%

Most reports have focused on only the venous thromboembolism risk, and few of them have studied specifically the association between hyperthyroidism and pulmonary embolism (PE).


nejmra1206531 96%

M ore than 6 million patients in the United States receive long-term anticoagulation therapy for the prevention of thromboembolism due to atrial fibrillation, placement of a mechanical heart-valve prosthesis, or venous thromboembolism.1 In addition, dual antiplatelet therapy (combination treatment with aspirin and a thienopyridine) after the placement of a coronary-artery stent has dramatically increased.


Circulation splanchnique 94%

Circulation splanchnique et états de choc Pr Pierre Asfar Laboratoire HIFIH Département de Réanimation Médicale CHU-Angers Analogy For Mixed Venous Oxygen Saturation Oxygen Uptake LOADING STATION CaO2 = (Hb .


Chamari 2001 90%

Mercier Venous blood lactate increase after vertical jumping in volleyball athletes Accepted:


LuminoTTTAJP 86%

A novel and direct prediction of this “humoral phototransduction” model is that light will stimulate the release by hemoglobin and production by hemoproteins of what today are described as the “gasotransmitters” carbon monoxide (CO) and nitric oxide (NO) in retinal venous blood.


TIH 85%

We place a high value on the prevention of venous limb gangrene and a low value on the cost of the additional days of the parental nonheparin anticoagulant.



puis pulmonaires (22) et digestifs (20) • Risque de MTEV si métastases (OR=19,8) vs cancer non métastasé • 18% des décès de patients cancéreux hospitalisés sont liés à une MTEV *Blom JW, Doggen CJ, Osanto S, Rosendaal FR (2005) Malignancies, prothrombotic mutations, and the risk of venous thrombosis.


noncirrhotic portal hypertension[1] 76%

The difference between the wedged hepatic venous pressure (WHVP) and the free hepatic venous pressure gives the hepatic venous pressure gradient (HVPG), which reflects portal pressure in cirrhotic patients (see the article by Groszmann and colleagues elsewhere in this issue).


Raper2016TrendsNeurosci 73%

CSF leaves the intracranial circulation by draining into the dural venous sinuses through arachnoid granulations, which contain valves that prevent the backflow of blood or CSF back into the CSF compartment [26–29] (Figure 1).


Clinicopathological features of nine cases of non-cirrhotic portal hypertension 71%

Stenoses were observed in some of the arterial branches in five cases and in some hepatic venous branches in four.


Pediatric Intracranial Hypertension 69%

However, those belonging to the SIH category were noted to have venous thrombosis in a significant minority.


Non cirrhotic portal hypertension versus idiopathic portal hypertension 67%

The common causes of NCPH include idiopathic portal hypertension (IPH), non-cirrhotic portal fibrosis (NCPF) and extrahepatic portal venous thrombosis (EHPVT).


33-11 61%

Hémodialyse veino-veineuse continue ou Continuous Veno Venous Hemodialysis CVVH :


An Uncommon Cause of 59%

Also noted, was a high estimated pulmonary pressure of 105 mmHg (central venous pressure measured with a central-vein catheter at 25 mmHg).


Peds 0617 Vascular Access 55%

In this issue, the indications and complications of peripheral intravenous access, intraosseous access, and central venous access are reviewed.



From hereon they will enter the capillaries and venous vessels which are roughly organized in a series of 4 arcades that end into the vena mesenteric superior, which itself becomes the vena porta.


Peds0715 Myocarditis-Pericarditis 50%

Obtain peripheral venous access in cases of suspected shock.


Acute Decomp Heart Failure 48%

Neck veins should be examined for the presence of jugular venous distension.


anatomy MCQ-BDS-2007 48%

d) Venous sinuses. e) Hassal’s corpuscles.


dossier de presse velo 46%

De la Baie du Mont-Saint-Michel à Annecy :


Article-PUGG-AVK-Drugs-and-Aging-Octobre-2013 45%

1 Introduction Oral anticoagulation therapy by vitamin K antagonists (VKA) has proven efficacy against thromboembolic disorders such as stroke prevention in atrial fibrillation (AF), venous thromboembolic disease (VTE), or heart valve replacement with mechanical prosthesis [1–3].


Étiologies du syndrome d’hypertension portale en milieu tropical 44%

Inclusion criteria We included in the study patients presenting at least one clinical sign of portal hypertension (ascitis, splenomegaly, collateral venous circulation), one endoscopic sign (esophageal and/or cardial varices, portal hypertension gastropathy), one ultrasonographic sign (dilatation of the portal and splenic veins, repermeabilisation of the umbilical vein) and who had been submitted to a liver biopsy.


Vascular Access in Pediatric Patients 44%

• The saphenous vein is the safest access point for venous cutdown in a pediatric patient.


Angus,NEJM,2013Review (1) 43%

or decrease in systolic pressure of >40 mm Hg in adults or to >2 SD below the lower limit of the normal range for age) Elevated mixed venous oxygen saturation (>70%)‡ Elevated cardiac index (>3.5 liters/min/square meter of body-surface area)§ Organ-dysfunction variables Arterial hypoxemia (ratio of the partial pressure of arterial oxygen to the fraction of inspired oxygen, <300) Acute oliguria (urine output, <0.5 ml/kg/hr or 45 ml/hr for at least 2 hr) Increase in creatinine level of >0.5 mg/dl (>44 μmol/liter) Coagulation abnormalities (international normalized ratio, >1.5;